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  • br Polybrominated diphenyl ethers occurrence dietary exposur

    2019-09-23


    Polybrominated diphenyl ethers: occurrence, dietary exposure, and toxicology.
    Environmental Working Group. In the dust. Toxic Fire Retardants in American Homes Washington, D.C.: Environmental Working Group; 2007 [Available from: http:// www.ewg.org/node/8452.
    Toxicological function of adipose tissue: focus on persistent organic pollutants.
    Lorber, M., 2008. Exposure of Americans to polybrominated diphenyl ethers. J. Expo. Sci.
    Epidemiologic evaluation of measurement data in the presence of detection limits.
    Mouly, T.A., Toms, L.L., 2016. Breast cancer and persistent organic pollutants (excluding DDT): a systematic literature review. Environ. Sci. Pollut. Res. Int. 23 (22), 22385–22407. National Cancer Institute (NCI) Surveillance Epidemiology and End Results (SEER) Cancer Stat Facts: Common Cancer Sites: National Cancer Institute (NCI) Surveillance, Epidemiology and End Results (SEER) 2013 [Available from: https://
    seer.cancer.gov/statfacts/html/common.html.
    Talsness, C.E., Kuriyama, S.N., Sterner-Kock, A., Schnitker, P., Grande, S.W., Shakibaei, M., et al., 2008. In utero and lactational exposures to low doses of polybrominated diphenyl ether-47 alter the Quizartinib (AC220) and thyroid gland of female rat offspring. Environ. Health Perspect. 116 (3), 308–314.
    U.S. Environmental Protection Agency. Toxicological review of decabromodiphenyl ether (BDE-209). In Support of Summary Information on the Integrated Risk Information System (IRIS) Washington, D.C.June 2008 [Available from: https://cfpub.epa.gov/ ncea/iris/search/index.cfm?keyword=BDE-209.
    U.S. Environmental Protection Agency (EPA). Technical fact sheet - polybrominated di-phenyl ethers (PBDEs), EPA 505-F-17-015 2017 [cited 2018. Available from: https:// www.epa.gov/sites/production/files/2014-03/documents/ffrrofactsheet_ contaminant_perchlorate_january2014_final_0.pdf.
    United States Environmental Protection Agency, 2008. Emerging contaminants – poly-brominated diphenyl ethers (PBDE) and polybrominated biphenyls (PBB) fact sheet. In: Solid Waste and Emergency Response, editor. US Department of Health and Human Services. DRAFT: NTP Technical Report on the the Toxiciology of A Pentabromodiphenyl Ether Mixture [DE-71 (Technical Grade)] 2015 Available from: https://ntp.niehs.nih.gov/ntp/about_ntp/trpanel/2015/june/tr589_ peerdraft.pdf.
    Vance, V., Mourtzakis, M., McCargar, L., Hanning, R., 2011. Weight gain in breast cancer survivors: prevalence, pattern and health consequences. Obes. Rev. 12 (4), 282–294. Verner, M.A., Charbonneau, M., Lopez-Carrillo, L., Haddad, S., 2008. Physiologically based pharmacokinetic modeling of persistent organic pollutants for lifetime ex-posure assessment: a new tool in breast cancer epidemiologic studies. Environ. Health Perspect. 116 (7), 886–892.
    Organochlorine exposures and breast cancer risk in New York City women. Environ.
    Improving organochlorine biomarker models for cancer research. Cancer Epidemiol.
    Wolff, M.S., Anderson, H.A., Britton, J.A., Rothman, N., 2007. Pharmacokinetic varia-bility and modern epidemiology–the example of dichlorodiphenyltrichloroethane, body mass index, and birth cohort. Cancer Epidemiol. Biomark. Prev. 16 (10), 1925–1930.
    Article
    A Cancer-Associated Missense Mutation in PP2A-Aa Increases Centrosome Clustering during Mitosis
    Noelle V. Antao, Marina Marcet-Ortega, Paolo Cifani, Alex Kentsis, Emily A. Foley
    [email protected]
    HIGHLIGHTS
    A heterozygous P179R mutation in PP2A-Aa decreases PP2A phosphatase assembly
    Intracellular targeting of PP2AB56a in mitosis is reduced in PP2A-
    PP2A-AaP179R/+ cells are more proficient at centrosome clustering
    Increased centrosome clustering is likely due to PP2A-Aa loss of function
    The Authors.
    Article
    A Cancer-Associated Missense Mutation in PP2A-Aa Increases Centrosome Clustering during Mitosis